Search results for "Neuregulin 1"

showing 10 items of 10 documents

Irreversible changes occurring in long-term denervated Schwann cells affect delayed nerve repair.

2017

OBJECTIVEMultiple factors may affect functional recovery after peripheral nerve injury, among them the lesion site and the interval between the injury and the surgical repair. When the nerve segment distal to the lesion site undergoes chronic degeneration, the ensuing regeneration (when allowed) is often poor. The aims of the current study were as follows: 1) to examine the expression changes of the neuregulin 1/ErbB system during long-term nerve degeneration; and 2) to investigate whether a chronically denervated distal nerve stump can sustain nerve regeneration of freshly axotomized axons.METHODSThis study used a rat surgical model of delayed nerve repair consisting of a cross suture betw…

0301 basic medicinePathologymedicine.medical_specialtyTime FactorsNerve rootNeuregulin-1Settore MED/19 - Chirurgia PlasticaSchwann cellNRG1/ErbB system03 medical and health sciences0302 clinical medicinePeripheral Nerve InjuriesMedicineAnimalsNeuregulin 1Rats Wistardelayed nerve repairDenervationneuregulin 1biologybusiness.industryRegeneration (biology)General MedicineAnatomyRecovery of FunctionDenervationMedian nerveNerve RegenerationRats030104 developmental biologymedicine.anatomical_structureperipheral nervePeripheral nerve injuryNerve Degenerationstereologybiology.proteinFemaleSchwann CellsbusinessEpineurial repair030217 neurology & neurosurgeryJournal of neurosurgery
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Preclinical and Clinical Characterization of Fibroblast-derived Neuregulin-1 on Trastuzumab and Pertuzumab Activity in HER2-positive Breast Cancer.

2021

[Purpose]: To characterize expression of neuregulin-1 (NRG1), an HER3 ligand, in HER2-positive breast cancer and its relation with the efficacy of trastuzumab with or without pertuzumab.

Cancer ResearchStromal cellReceptor ErbB-2medicine.medical_treatmentNeuregulin-1Drug Evaluation PreclinicalBreast NeoplasmsAntibodies Monoclonal HumanizedBreast cancerAntineoplastic Agents ImmunologicalTrastuzumabAntineoplastic Combined Chemotherapy Protocolsmental disordersmedicineTumor Cells CulturedHumansNeuregulin 1skin and connective tissue diseasesneoplasmsNeoadjuvant therapyRetrospective Studiesbiologybusiness.industryFibroblastsTrastuzumabmedicine.diseasebody regionsTreatment OutcomeOncologyCancer cellbiology.proteinCancer researchImmunohistochemistryFemalePertuzumabbusinessmedicine.drug
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Nrg1 haploinsufficiency alters inhibitory cortical circuits

2021

Neuregulin 1 (NRG1) and its receptor ERBB4 are schizophrenia (SZ) risk genes that control the development of both excitatory and inhibitory cortical circuits. Most studies focused on the characterization ErbB4 deficient mice. However, ErbB4 deletion concurrently perturbs the signaling of Nrg1 and Neuregulin 3 (Nrg3), another ligand expressed in the cortex. In addition, NRG1 polymorphisms linked to SZ locate mainly in non-coding regions and they may partially reduce Nrg1 expression. Here, to study the relevance of Nrg1 partial loss-of-function in cortical circuits we characterized a recently developed haploinsufficient mouse model of Nrg1 (Nrg1tm1Lex). These mice display SZ-like behavioral d…

Cortical neuronsReceptor ErbB-4Neuregulin-1Gene ExpressionneuronsNeurosciences. Biological psychiatry. NeuropsychiatryHaploinsufficiencyBiologyInhibitory postsynaptic potentialHippocampusMagnetic&nbspMiceInterneuronsNeuregulin 3mental disordersMagnetic resonance spectroscopyAnimalsRNA MessengerneurotransmissionNeuregulin 1GABAergic Neuronsgamma-Aminobutyric AcidInhibitory&nbspCerebral CortexNrg1resonance spectroscopyNeural InhibitionMagnetic Resonance ImagingCortex (botany)Inhibitory neurotransmissionParvalbuminsNeurologyInhibitory Postsynaptic PotentialsCalbindin 2Vesicular Glutamate Transport Protein 1biology.proteinExcitatory postsynaptic potentialSchizophreniaCalretininHaploinsufficiencyCortical&nbspNeuroscienceParvalbuminRC321-571Neurobiology of Disease
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Gene expression of neuregulin-1 isoforms in different brain regions of elderly schizophrenia patients

2010

One important risk gene in schizophrenia is neuregulin-1 (NRG1), which is expressed in different isoforms in the brain. To determine if alterations of NRG1 are present in schizophrenia, we measured gene expression of NRG1 and its main isoforms as well as the impact of genetic variation of NRG1 in an exploratory study examining three brain regions instead of only one as published so far. In all, we examined post-mortem samples from 11 schizophrenia patients and eight normal subjects. We investigated gene expression of total NRG1 and isoforms I, II and III by real-time PCR in the prefrontal cortex (Brodmann areas 9 and 10) and right hippocampal tissue. For the genetic study, we genotyped the …

Gene isoformMalemedicine.medical_specialtyGenotypeNeuregulin-1HippocampusGene ExpressionPrefrontal CortexHippocampal formationHippocampusPolymorphism Single Nucleotide03 medical and health sciences0302 clinical medicineInternal medicinemental disordersGenetic variationGene expressionmedicineHumansProtein IsoformsNeuregulin 1Prefrontal cortexAllelesBiological Psychiatry030304 developmental biologyAged0303 health sciencesbiologyReverse Transcriptase Polymerase Chain ReactionBrainmedicine.disease030227 psychiatryPsychiatry and Mental healthEndocrinologyHaplotypesSchizophreniabiology.proteinSchizophreniaFemalePsychologyNeuroscience030217 neurology & neurosurgeryWorld Journal of Biological Psychiatry
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Dysregulated Expression of Neuregulin-1 by Cortical Pyramidal Neurons Disrupts Synaptic Plasticity

2014

Summary Neuregulin-1 ( NRG1 ) gene variants are associated with increased genetic risk for schizophrenia. It is unclear whether risk haplotypes cause elevated or decreased expression of NRG1 in the brains of schizophrenia patients, given that both findings have been reported from autopsy studies. To study NRG1 functions in vivo, we generated mouse mutants with reduced and elevated NRG1 levels and analyzed the impact on cortical functions. Loss of NRG1 from cortical projection neurons resulted in increased inhibitory neurotransmission, reduced synaptic plasticity, and hypoactivity. Neuronal overexpression of cysteine-rich domain (CRD)-NRG1, the major brain isoform, caused unbalanced excitato…

MaleDendritic SpinesNeuregulin-1Nonsynaptic plasticityGene ExpressionMice TransgenicNeurotransmissionInhibitory postsynaptic potentialSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyCell MovementInterneuronsConditioning Psychologicalmental disordersAnimalsNeuregulin 1lcsh:QH301-705.5CA1 Region HippocampalNeuronal PlasticitybiologyPyramidal CellsAnatomyFearCortex (botany)Synaptic fatiguelcsh:Biology (General)Synaptic plasticitybiology.proteinExcitatory postsynaptic potentialFemaleNerve NetNeuroscience
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Integrative proteomics: functional and molecular characterization of a particular glutamate-related neuregulin isoform.

2005

Glutamate is the major excitatory neurotransmitter in the mammalian brain and is related to memory by calcium-conducting receptors. Neuregulins have emerged as long-term modulating molecules of synaptic signaling by glutamate receptors, playing a role in some cognition/memory-related disorders and moreover being part of transient functional microdomains, called lipid rafts. Here we characterize one specific isoform of neuregulin as a central biomarker for glutamate-related signaling, integrating results from in vitro and in vivo models by a differential functional and proteomic approach.

ProteomicsNeuregulin-1Glutamic AcidNerve Tissue ProteinsBiochemistryHippocampusRats Sprague-DawleyAlzheimer DiseaseAnimalsHumansLearningProtein IsoformsNeuregulin 1ReceptorLipid raftCells CulturedbiologyGlutamate receptorGeneral ChemistryGlutamic acidCell biologyRatsbiology.proteinNeuregulinCalciumFemaleSynaptic signalingSignal transductionBiomarkersSignal TransductionJournal of proteome research
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ErbB-3 activation by NRG-1β sustains growth and promotes vemurafenib resistance in BRAF-V600E colon cancer stem cells (CSCs)

2015

Approximately 5-10% of metastatic colorectal cancers harbor a BRAF-V600E mutation, which is correlated with resistance to EGFR-targeted therapies and worse clinical outcome. Vice versa, targeted inhibition of BRAF-V600E with the selective inhibitor PLX 4032 (Vemurafenib) is severely limited due to feedback re-activation of EGFR in these tumors. Mounting evidence indicates that upregulation of the ErbB-3 signaling axis may occur in response to several targeted therapeutics, including Vemurafenib, and NRG-1β-dependent re-activation of the PI3K/AKT survival pathway has been associated with therapy resistance. Here we show that colon CSCs express, next to EGFR and ErbB-2, also significant amoun…

Proto-Oncogene Proteins B-rafMAPK/ERK pathwayIndolesReceptor ErbB-3Colorectal cancerNeuregulin-1colon cancer stem cellsMice NudeAntineoplastic AgentsMiceErbBErbB-3medicineAnimalsHumansNeuregulin 1VemurafenibClonogenic assayskin and connective tissue diseasesProtein kinase BneoplasmsPI3K/AKT/mTOR pathwayCell ProliferationOligonucleotide Array Sequence AnalysisNRG-1βSulfonamidesbiologyReverse Transcriptase Polymerase Chain Reactionbusiness.industryFlow Cytometrymedicine.diseaseImmunohistochemistryXenograft Model Antitumor AssaysVemurafenibOncologyDrug Resistance NeoplasmColonic NeoplasmsImmunologyNeoplastic Stem CellsCancer researchbiology.proteinbusinessPriority Research Papermedicine.drug
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Disturbed structural connectivity in schizophrenia primary factor in pathology or epiphenomenon?

2007

Indirect evidence for disturbed structural connectivity of subcortical fiber tracts in schizophrenia has been obtained from functional neuroimaging and electrophysiologic studies. During the past few years, new structural imaging methods have become available. Diffusion tensor imaging and magnetization transfer imaging (MTI) have been used to investigate directly whether fiber tract abnormalities are indeed present in schizophrenia. To date, findings are inconsistent that may express problems related to methodological issues and sample size. Also, pathological processes detectable with these new techniques are not yet well understood. Nevertheless, with growing evidence of disturbed structu…

PsychosisPathologymedicine.medical_specialtySchizophrenia (object-oriented programming)Neuregulin-1EpiphenomenonNerve Tissue ProteinsMyelinNerve FibersFunctional neuroimagingmental disordersmedicineHumansNeuregulin 1Myelin SheathbiologyGenes erbBmedicine.diseaseMagnetic Resonance ImagingOligodendrocytePsychiatry and Mental healthOligodendrogliamedicine.anatomical_structureDiffusion Magnetic Resonance Imagingbiology.proteinSchizophreniaNeuregulinNerve NetPsychologyNeuroscienceRegular ArticlesSchizophrenia bulletin
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229 – ERBB4 genotype effects on human brain structure

2008

Background:Disturbed functional and structural brain connectivity in schizophrenia has been shown in a large number of studies. There is evidence from several neuroimaging and post mortem studies that altered neuronal myelination may in part account for this deficit. Recent investigations have suggested that variations of the genes that encode the Neuregulin 1 (NRG1) ErbB4 receptor complex might be associated with schizophrenia illness. As NRG1 and ErbB4 have been implicated in myelination and neuronal proliferation. We investigated whether cerebral micro and macrostructure is predicted by two risk haplotypes of the ErbB4 gene. Methods: The effects of two single-nucleotide polymorphisms (SN…

Receptor complexHaplotypeHuman brainBiologymedicine.diseaseWhite matterPsychiatry and Mental healthmedicine.anatomical_structurenervous systemSchizophreniaFractional anisotropybiology.proteinmedicineNeuregulin 1NeuroscienceBiological PsychiatryERBB4Schizophrenia Research
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The effects of α-secretase ADAM10 on the proteolysis of neuregulin-1

2009

Although ADAM10 is a major alpha-secretase involved in non-amyloidogenic processing of the amyloid precursor protein, several additional substrates have been identified, most of them in vitro. Thus, therapeutical approaches for the prevention of Alzheimer's disease by upregulation of this metalloproteinase may have severe side effects. In the present study, we examined whether the ErbB receptor ligand neuregulin-1, which is essential for myelination and other important neuronal functions, is cleaved by ADAM10. Studies with beta- and gamma-secretase inhibitors, as well as with the metalloproteinase inhibitor GM6001, revealed an inhibition of neuregulin-1 processing in human astroglioma cell …

biologyADAM10Cell BiologySheddaseBiochemistryDownregulation and upregulationErbBIn vivoAmyloid precursor proteinbiology.proteinCancer researchAstrogliomaNeuregulin 1Molecular BiologyFEBS Journal
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